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Ozone Therapy for the Treatment of Herniated Intervertebral Discs (HID)

by Gaetano Morello, BSc., N.D.

Introduction:

Low back pain is one of the most common complaints during primary care office visits. It is estimated that approximately 80% of all Americans report low back problems at some time in their lives.[1] Among working age people, as many as 20 percent experience back symptoms at least every year. In the United States, spinal diseases are the most common cause of disability in persons under the age of 45.[2] What is even more surprising is that the estimated costs of direct medical and indirect expenses for low back pain in the U.S. range from $20 to $100 billion annually. Spine care results in expenditures two to three time greater than cardiac services for many health plans.[3]

Some of the main causes of back pain include spinal stenosis, sciatica, muscle strain, bulging or herniated discs and degenerative disc disease. Herniated intervertebral discs (HID) are the most common cause of low back pain associated with a defined structural abnormality.[4,5]

The most common treatments for HID include surgical discectomy, epidural steroid injection and the use of non-steroidal anti- inflammatory drugs (NSAIDs). Other therapies have included chemonucleolysis, prolotherapy, traction and conservative therapy (rest). [6, 7-9]

Patients who are not helped by weeks of conservative therapy are often referred for surgery on the premise that further non-operative care is unlikely to help. More recent data however, is suggesting that extended conservative therapy (up to 4 years) leads to favorable outcomes just as often as surgery. But it is this long period of time needed with conservative therapy that often makes surgery the treatment of choice.[10]

In the last 10 years there has been a growing interest in alternatives to surgery. Chemonucleolysis gained some favor in the early 80's and more recently prolotherapy has been receiving some much-deserved attention.

Another alternative therapy that has been receiving exposure in Europe is the use of medical ozone (02/03 mixture) in the treatment of HID. Medical ozone has been widely used in Europe for 40 years and now is seriously being looked at as a possible treatment for herniated discs and other types of lower back pain. In order to acquire an understanding about the biochemical action of ozone in treating HID, a foundation of the structure of the intervertebral disc and diagnostic criteria of HID is essential.[11,12]

Structure of the intervertebral disc:

The intervertebral discs make up about a quarter of the length of the vertebral column. They may be regarded as semielastic discs, which lie between vertebral bodies. Their physical characteristics permit them to serve as shock absorbers when the load on the vertebral column is suddenly increased.

Each disc consists of a peripheral part, the annulus fibrosus and a central part, the nucleus pulposus.[4] The annulus fibrosus is composed of fibrocartilage in which the collagen fibers are arranged in concentric lamellae that surround the nucleus pulposus. The lamellae are thick in the anterior and lateral portions of the anulus, but posteriorly are thinner and more densely packed. Water makes up about 60-70% of the weight of the annulus fibrosus while collagen and proteoglycans make up 50% and 20% respectively of the dry weight of the annulus.

The nucleus pulposus is gelatinous in structure and is the actual shock absorber between the vertebrae. Bio-mechanically, the fluid nature of the nucleus allows it to be deformed under pressure, but as a fluid, its volume cannot be compressed. When subjected to pressure from any given direction, the nucleus will attempt to deform and transmit the applied pressure in all directions. Water makes up about 70-90% of the nucleus pulposus although the exact fraction varies with age. The next major components are proteoglycans; the water of the nucleus is contained within the domain of these proteoglycans. The upper and lower surfaces of the bodies of adjacent vertebrae that abut onto the disc are covered with thin plate of hyaline cartilage (known as the vertebral end-plates).[13]

Diagnostic Considerations:

Diagnosis of HID comes from a thorough history, physical exam (PE), neurological and orthopedic tests, and is confirmed with proper diagnostic imaging.[4] The symptoms of HID include pain that can develop gradually or suddenly. It is worst on movement and may be exacerbated by the Valsalva maneuver. Paresthesias or numbness in the sensory distribution of the root may occur and tendon reflexes may be depressed. Straight leg raise may produce back pain because it stretches the nerve roots. Muscles supplied by the impaired root eventually become weak, wasted, flaccid and may show fasciculations.

Signs and symptoms of herniated discs are caused by compression of either nerve roots or the spinal cord. The specific signs and symptoms are based on which part is compressed and at which level the neural structures are compressed. The diagnosis of a herniated disc is usually ascertained from the characteristic clinical symptoms and diagnostic findings. Because more than 95% of lumbar disc herniations are at the L4-L5, L5-S1 levels, the physical examination should focus on abnormalities of the L5 and S1 nerve roots.

Neurological and orthopedic tests should include: Bowstrings sign (excellent for nerve root compression), straight leg raise (Lasegue test, for L5 and S1 radiculopathy), femoral stretch test (L4 radiculopathy), Achilles reflex (S1 nerve root) and Patellar reflex (L4 nerve root). Plain radiography is not useful in diagnosis of a HID but computed tomography (CT) or magnetic resonance imaging (MRI) is often diagnostic. MRI is becoming the preferred choice of diagnostic imaging because of the lack of radiation and the superior view of the discs.[4,7,13,11]

Ozone Therapy

The use of medical ozone in the treatment of HID was developed by Cesare Verga (an orthopedic surgeon) in 1983. Dr. Verga has since treated over 8,000 patients with HID and documented a 95% cure rate (35% of these cases had at least one prior surgical intervention). In 80% of the clinically healed cases there are also corresponding CT and MRI images confirming resolution. In 15 years of treatments Dr. Verga states that relapses occur in less than 2% of cases. The method he uses which we will term the "Verga Method" (VM), involves the administration of 40/60 cc of ozone gas (O2-O3 mixture) at a concentration of 20/30 micrograms per cc, repeated 8-14 times. The injection is generally made into the paravertebral musculature, and in the hernia zone. The injection itself, except for a slight sense of localized pain of short duration, is generally painless and well tolerated. There have been a number of European studies confirming the efficacy of the "Verga Method" and new studies are currently underway.[11,15-17]

How does Ozone Therapy using the "Verga Method" work? Although the exact mechanism of action is not completely understood, there are a number of characteristics of medical ozone that offer some insights into its mode of action in treating HID. Thus, it has been hypothesized that the success of the "Verga Method" is based on four main biochemical actions on the intervertebral disc and its surrounding tissues. The various proposed mechanisms of action are:

1. Several studies suggest disc inflammation as a mechanism of sciatica due to disc herniation.[7,18] Ozone has been shown to have an effect on the inflammatory cascade by altering the breakdown of arachidonic acid to inflammatory prostaglandins.[11] Therefore, by reducing the inflammatory components there is a corresponding reduction in pain.

2. Another hypothesis as to the mechanism of action of the VM is based on the fact that the circulatory environment around a HID can be compromised. The herniation can impinge on the venous and arterial flow and cause phlebostasis and arteriostenosis. The combination of these two conditions can then lead to a serious hypoxemia. These factors can contribute to the pain experienced with a HID. In fact, the sensory roots more so than the motor are particularly sensitive to anoxia. By applying the ozone to HID area, both a direct and indirect hyperoxygenation of the zone occurs, which reduces the pain. The direct effect is the oxygen directly diffusing into the area. The indirect action is the ozone causing an increase in 2,3-DPG (diphosphoglycerate) which has a direct effect in the release of O2 from hemoglobin.[12] The end result is an increase in the amount of oxygen and a reduction in anoxia.

3. The third plausible mechanism of action is the direct effect of the ozone on the herniation. It is well established that the nucleus pulposus (the actual part of the disc that herniates) is 70-90% water contained within the domain of proteoglycans.[13] The water binding capacity of the proteoglycan molecule is partially a property of its size and physical shape, but the main force that holds water to the molecule stems from the ionic, carboxyl (COOH) and sulphate (SO4) radicals of the glycosaminoglycan chains. The ozone can have a direct effect on these carboxyl and sulphate groups, breaking down some of these glycosaminoglycan chains which make up the proteoglycans. The destruction of these cross-linked structures reduces their ability to hold water therefore diminishing the size of the herniation. [12,15,16]

4. Another action which may prove to be one of the most important is the stimulation of fibroblastic activity by ozone. Fibroblasts initiate the repair process by stimulating the deposition of collagen. Although yet to be validated, this mode of action could explain the resolution of HID on CT scans and the small percentage of patients who have relapses after the completion of treatment plan. [12,16]

How is the Verga Method performed?

The VM is performed by injecting 60 cc of O2-O3 mixture in 4 divided doses paravertebrally around the HID zone. Each site injected will receive approximately 15 cc of the gas. The concentration of ozone used will be between 20-30 micrograms/cc. A glass syringe or a silicone coated disposable syringe must be used in order to properly administer the ozone mixture (needles used are 22-25 gauge, 1.5-2.0 inches in length). The amount of treatments needed varies from patient to patient; but the average patient has recovery in about 14 sessions.

Advantages of the Verga Method:

1. A great advantage of this method is that it practically has no contraindications. 2. There are no limits to the number of sessions, adapting to the necessity of the patient. 3. Patients who have already had back surgery even several times have benefited from the VM. 4. The mode in which the VM works, does not in anyway alter the biomechanics of the back. 5. It does not require hospitalization.

Final Comments

The Verga Method has shown to be a promising therapy for patients with herniated intervertebral discs. Its modes of action seem to address the underlying pathology without disturbing the biomechanics of the back.. There is a definite need for more clinical research to fully understand the promising results this method is providing physicians. But the clinical results obtained thus far by a large number of orthopedic surgeons in Europe offer real hope to people suffering from this debilitating condition.

References:

1.Damkot DK, Pope MII, Lord J, Frymoyer JW. The relationship between work history, work environment and low-back pain in men. Spine. 1984;9:395-399
2.Anderson GBJ. Epidemiology of spinal disorders. In: Frymoyer JW, ed. The Adult Spine. New York, NY: Raven Press; 1991: 107-146
3.Snook SH. The costs of back pain in industry. Occup Med.;3:1, 1988
4.Wyngaarden James B., Smith Lloyd H.: Cecil Textbook of Medicine.18th Edition; W.B. Saunders Company, 1988
5.Frymoyer JW. Back pain and sciatic. N. Engl. J. Med. 318:291-300, 1988
6.Bell GR, Rothman RH. The conservative treatment of sciatica. Spine, 9:54-56, 1984
7.Scheer SJ et al.: Randomized controlled trials in industrial low back pain relating to return to work. Part 2. Discogenic Low Back Pain. Arch Phys Med Rehabil 77:1189-1197, 1996
8.Herkowitz HN. Current status of percutaneous discectomy and chemonucleolysis. Orthop Clin North Am 22:327-32, 1991
9.Carette S et al.: Epidural corticosteroid injections for sciatica due to herniated nucleus pulposus. N Engl J Med 336:1634-40, 1997
10.Deyo RA et al.: How many days of bed rest for acute low back pain, a randomized clinical trial. N Engl J Med, 315:1064-1070, 1986
11.Iliakis E et al.: Ozonetherapy in the treatment of low back pain. Orthopaedics 8, 1:29-33, 1995
12.Viebahn R. The Use of Ozone in Medicine. 2nd Edition; Karl Haug Publishers, 1994
13.Bogduk Nikolai: The inter-body joint and intervertebral discs. In: Clinical Anatomy of the Lumbar Spine and Sacrum. Third edition; New York: Churchill Livingstone, 1997
14.Mazion JM: Illustrated Manual of Neurological (Reflexes/signs/ tests) and Orthopedic(signs/tests/maneuvers) for Office Procedures. 2nd edition; Phoenix: JM Mazion, 1980
15.Iliakis E. Utilization of Ozonetherapy in the practice of orthopedics. Acta Toxicol Ther, 17, (2-3) 249-251, 1996
16.Sforza A, Sforza G. Confirmation of the effectiveness of local percutaneous injections of ozone in chronic and acute radiculopathy. Acta Toxicol Ther, 17 (2-3) 245-248, 1996
17.Gualandi G, et al.: Preliminary trials, with ozone therapy in the treatment low back pain. Acta Toxicol Ther, 17, (2-3) 261-264, 1996
18.Piperno M et al.: Phospholipase A2 activity in herniated lumbar discs clinical correlations and inhibition by piroxicam. Spine, 22: 2061-2065, 1997



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